RESILIENCE Bundle
The RESILIENCE Bundle is a three-vial cellular-resilience research stack: NAD+ 500 mg, MOTS-C 10 mg and BPC-157 10 mg — at 10% off the individual vial prices.
NAD+ is a coenzyme central to cellular energy metabolism, whose roles during ageing have been comprehensively reviewed[1]. MOTS-C is a mitochondrial-derived peptide that promoted metabolic homeostasis in mouse models[3]. BPC-157 is a stable gastric pentadecapeptide studied in tendon-healing models[5].
Each vial is supplied as a lyophilized powder for reconstitution, and each compound has its own lot-specific third-party certificate of analysis, available on request. Findings above come from preclinical (animal and cell-based) research and review literature on these molecules and are provided as scientific reference for laboratory research only.
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Cellular Resilience Stack
Three Vials, One Research Focus
The RESILIENCE Bundle is built around cellular-resilience research. NAD+ is the coenzyme at the centre of cellular energy metabolism, whose roles during ageing have been comprehensively reviewed[1]. MOTS-C is a mitochondrial-derived peptide that promoted metabolic homeostasis in preclinical models[3]. BPC-157 adds the repair angle: a stable gastric pentadecapeptide documented in tendon-healing models[5].
- Cellular energy, mitochondrial and repair research in one set
- NAD+ — coenzyme of cellular energy metabolism
- MOTS-C — mitochondrial-derived peptide research
- BPC-157 — tendon-healing research models
- Lot-specific third-party COA for each compound
Inside the Bundle
What's in the Stack
The bundle ships as three separate lyophilized vials: NAD+ 500 mg, MOTS-C 10 mg and BPC-157 10 mg. The three compounds cover complementary layers of cellular research: coenzyme biochemistry — declining NAD+ levels have been studied in ageing and metabolism research[2] — mitochondrial signalling, where MOTS-c was described as an exercise-induced regulator of muscle homeostasis in mice[4], and peptide repair signalling (BPC-157[5]).
- NAD+ — 500 mg lyophilized vial
- MOTS-C — 10 mg lyophilized vial
- BPC-157 — 10 mg lyophilized vial
- Coenzyme + mitochondrial + repair research coverage
- 10% saving versus buying the three vials separately
Research Applications
Areas of Scientific Investigation
Each compound carries its own line of evidence: NAD+ metabolism and its roles during ageing are the subject of a major review literature[1]; in mouse models, MOTS-c promoted metabolic homeostasis and reduced obesity and insulin resistance[3]; and in a rat model, BPC-157 accelerated healing of the transected Achilles tendon[6]. No published study evaluates the three compounds in combination; researchers using them side by side are combining independent lines of evidence.
- Cellular energy and coenzyme metabolism research
- Mitochondrial-peptide signalling studies
- Ageing and metabolic-homeostasis models
- Tendon and tissue-repair research models
Understanding the Resilience Stack: Mechanism of Action
NAD+ (nicotinamide adenine dinucleotide) is a coenzyme central to cellular energy metabolism and a required substrate for sirtuins and PARP enzymes. Its metabolism and roles in cellular processes during ageing have been comprehensively reviewed[1], and declining NAD+ levels have been studied in ageing, metabolism and neurodegeneration research[2].
MOTS-c is a 16-amino-acid peptide encoded within the mitochondrial genome. In preclinical models it promoted metabolic homeostasis and reduced obesity and insulin resistance in mice[3], and was described as an exercise-induced, mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis in mice[4].
BPC-157 is a 15-amino-acid fragment of the Body Protection Compound found in gastric juice. In preclinical models it has been reported to promote tissue-repair signalling: it promoted tendon-fibroblast outgrowth and migration, likely via the FAK–paxillin pathway[5], and accelerated healing of transected tendon in a rat study[6].
Together, the three vials give researchers a cellular-resilience toolkit: coenzyme biochemistry (NAD+), mitochondrial-peptide signalling (MOTS-c) and peptide repair signalling (BPC-157). Each is supported by its own line of preclinical evidence; no published study evaluates the combination itself. These materials are supplied strictly for laboratory research use.
Scientific Literature
- Covarrubias AJ, Perrone R, Grozio A, Verdin E. NAD+ metabolism and its roles in cellular processes during ageing. Nat Rev Mol Cell Biol. 2021;22(2):119-141.
- Verdin E. NAD+ in aging, metabolism, and neurodegeneration. Science. 2015;350(6265):1208-13.
- Lee C, Zeng J, Drew BG, et al. The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance. Cell Metab. 2015;21(3):443-454.
- Reynolds JC, Lai RW, Woodhead JST, et al. MOTS-c is an exercise-induced mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis. Nat Commun. 2021;12(1):470.
- Chang CH, Tsai WC, Lin MS, Hsu YH, Pang JH. The promoting effect of pentadecapeptide BPC 157 on tendon healing involves tendon outgrowth, cell survival, and cell migration. J Appl Physiol (1985). 2011;110(3):774-80.
- Staresinic M, Sebecic B, Patrlj L, et al. Gastric pentadecapeptide BPC 157 accelerates healing of transected rat Achilles tendon and in vitro stimulates tendocytes growth. J Orthop Res. 2003;21(6):976-83.
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RESILIENCE Bundle
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$189.00$340.00
- ≥99%96%
- 3 vials3 vials
- Free 2-Day Shipping3-10 Business Day
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